Help with facts.

Tuesday night my mother picked me up from a friends and asked if i've been smoking pot again, and i didn't feel i needed to lie so i told her i have been. Me and her have a pretty good relationship, and shes still pretty young.
(shes 34 and im 17) She replied with the usual, ''glad you're being honest, but i really don't want you to smoke." I asked her why and she gave me her reasons and i explained that i wouldn't let it become a problem, i wouldn't be stealing from anyone, i wouldn't be driving under the influence, basically doing it responsibly inside a safe house. Basically she believes all the anti-weed propoganda bullshit. Shes uneducated and i wanted to pull up a bunch of facts that actually seem believable instead of some 'pot head site that feeds lies', as she says. I'm not asking you guys to do all the work, but if you could contribute any good sites as im searching, it would be appreciated. Thanks :cool:


p.s. That thread i made a bit ago about her getting a med card, she told me she was joking :-/

Heart Problems
It is accepted in medical circles today that marijuana use causes no evident long-term cardiovascular problems for normal persons. Marijuana smoking, however, does cause changes in the heart and body's circulation characteristic of stress, which may complicate preexisting cardiovascular problems like hypertension, cerebrovascular disease, and coronary atherosclerosis. Marijuana's effects on blood pressure are complex and inconsistent as of yet.
Hormones
Chronic marijuana use has not been found to alter testosterone or other sex hormone levels. In contrast, heavy alcohol use is known to lower these same testosterone levels.

Reproductive Damage
No trustworthy study has ever shown that marijuana use damages the reproductive system, or causes chromosome breakage. Studies of actual human populations have failed to demonstrate that marijuana adversely affects the reproductive system. Claims that marijuana use may impair hormone production, menstrual cycles, or fertility in females are both unproven and unfounded.

The Immune System
Studies in which lab rats were injected with extremely large quantities of THC (the active compound in marijuana) have found that marijuana (in such unrealistically huge quantities) does have an "immunosuppressive effect" in those lab rats, in that it temporarily shuts off certain cells in the liver called lymphocytes and macrophages. These macrophages are useful in fighting off bacterial, not viral, infections.

But this is only for the duration of intoxication. There also exists some evidence that marijuana metabolites remain in the lungs for up to seven months after smoking has ceased, possibly affecting the immune system of the lungs (but not by turning the cells off).

This said, doctors and researchers are still not sure that the immune system is actually negatively affected in realistic situations since there are no numbers to support the idea. In fact, three studies showed that THC might have actually stimulated the immune system in the people studied.

Cancer
Smoking marijuana has the potential to cause both bronchitis and cancer of the lungs, throat, and neck, but this is generally no different than inhaling any other burnt carbon-containing matter since they all increase the number of lesions (and therefore possible infections) in your airways.

The Gateway Effect
Marijuana use has not been found to act as a gateway drug to the use of harder drugs. Studies show that when the Dutch partially legalized marijuana in the 70's, heroin and cocaine use substantially declined, despite a slight increase in marijuana use.

If the stepping stone theory were true, use should have gone up rather than down. In reality, it appears that marijuana use tends to substitute for the use of relatively more dangerous hard drugs like cocaine and heroin, rather than lead to their use.

Thus, oftentimes strict marijuana laws themselves are the most significant factor involved in moving on to harder drugs like cocaine. Such is the case in Nevada and Arizona, the states toughest on marijuana use.

Thanks for the help and support.:jointsmile:

search for a post i made called TRUTH - True Marijuana Facts


loooots of info

Will do, thanks.

one of the anti pot smoking commercials admit pot is safe,, remember, "we smoke weed, we dont die, we dont kill people, we dont do crazy stuff, all we do is sit on pete's couch for 14 hours, "

Yeah i saw that, but my moms brother is like 50 and he still lives with his mom and she doesn't want me doing that either.

Have her watch the 1939 reefer madness, it is so ridiculous she will definitely be open to the truth. And perhaps a med mj card as well. :thumbsup:

Quote:

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Originally Posted by Mean Green Charlene

Yeah i saw that, but my moms brother is like 50 and he still lives with his mom and she doesn't want me doing that either.

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lol, goddamn,, thats unusual,, no disrespect but thats pretty bumish ,

BUMISH- a noun i made up that is used to describe someone that has traits of a bum,
also could be used as an adjective

Yeah she doesn't want me sitting around doing nothing with my life, she tried using that against me. I'm a great debater though, & pretty educated on the subject. No way she can win ;]

Marijuana Smoking Does Not Cause Lung Cancer, UCLA Expert Dr. Tashkin Concludes
Protective Effect "Not Unreasonable"


Report by Fred Gardner - C-Notes June 29, 2005
Marijuana smoking -"even heavy longterm use"- does not cause cancer of the lung, upper airways, or esophagus, Dr. Donald Tashkin reported at this year's meeting of the International Cannabinoid Research Society.

Coming from Tashkin, this conclusion had extra significance for the assembled drug-company and university-based scientists (most of whom get funding from the U.S. National Institute on Drug Abuse). Over the years, Tashkin's lab at UCLA has produced irrefutable evidence of the damage that marijuana smoke wreaks on bronchial tissue. With NIDA's support, Tashkin and colleagues have identified the potent carcinogens in marijuana smoke, biopsied and made photomicrographs of pre-malignant cells, and studied the molecular changes occurring within them. It is Tashkin's research that the Drug Czar's office cites in ads linking marijuana to lung cancer. Tashkin himself has long believed in a causal relationship, despite a study in which Stephen Sidney examined the files of 64,000 Kaiser patients and found that marijuana users didn't develop lung cancer at a higher rate or die earlier than non-users. Of five smaller studies on the question, only two -involving a total of about 300 patients- concluded that marijuana smoking causes lung cancer. Tashkin decided to settle the question by conducting a large, population-based, case-controlled study."Our major hypothesis," he told the ICRS, "was that heavy, longterm use of marijuana will increase the risk of lung and upper-airways cancers."

The Los Angeles County Cancer Surveillance program provided Tashkin's team with the names of 1,209 L.A. residents aged 59 or younger with cancer (611 lung, 403 oral/pharyngeal, 90 laryngeal, 108 esophageal). Interviewers collected extensive lifetime histories of marijuana, tobacco, alcohol and other drug use, and data on diet, occupational exposures, family history of cancer, and various "socio-demographic factors." Exposure to marijuana was measured in joint years (joints per day x years that number smoked). Controls were found based on age, gender and neighborhood. Among them, 46% had never used marijuana, 31% had used for less than one joint year, 12% had used for 1-10 j-yrs, 5% had used 10-30 j-yrs, 2% had used for 30-60 j-yrs, and 3% had used for more than 60 j-yrs. Tashkin controlled for tobacco use and calculated the relative risk of marijuana use resulting in lung and upper airways cancers. A relative risk ratio of .72 means that for every 100 non-users who get lung cancer, only 72 people who smoke get lung cancer. All the odds ratios in Tashkinπs study turned out to be less than one! Compared with subjects who had used less than one joint year, the estimated odds ratios for lung cancer were .78 for 1-10 j-yrs [according to the abstract book and .66 according to notes from the talk] .74 for 10-30 j-yrs; .85 for 30-60 j-yrs; and 0.81 for more than 60 j-yrs. The estimated odds ratios for oral/pharyngeal cancers were 0.92 for 1-10 j-yrs; 0.89 for 10-30 j-yrs; 0.81 for 30-60 j-yrs; and 1.0 for more than 60 j-yrs. "Similar, though less precise results were obtained for the other cancer sites," Tashkin reported. "We found absolutely no suggestion of a dose response." The data on tobacco use, as expected, revealed "a very potent effect and a clear dose-response relationship -a 21-fold greater risk of developing lung cancer if you smoke more than two packs a day." Similarly high odds obtained for oral/pharyngeal cancer, laryngeal cancer and esophageal cancer. "So, in summary" Tashkin concluded, "we failed to observe a positive association of marijuana use and other potential confounders."

There was time for only one question, said the moderator, and San Francisco oncologist Donald Abrams, M.D., was already at the microphone: "You don't see any positive correlation, but in at least one category, it almost looked like there was a negative correlation, i.e., a protective effect. Could you comment on that?" [Abrams was referring to Tashkinπs lung-cancer-only data for marijuana-only smokers in 1-10 j-yrs category.] "Yes," said Tashkin. "The odds ratios are less than one almost consistently, and in one category that relationship was significant, but I think that it would be difficult to extract from these data the conclusion that marijuana is protective against lung cancer. But that is not an unreasonable hypothesis."