THE TRUTH ABOUT MY THE WEED!

i founf this posted on www.hightimes.com


http://www.hightimes.com/ht/news/con...id=1114&aid=10

i boled a few things that i though where important.



Scientific American: The Brain's Own Marijuana
2004-12-20 >> news category >> general
Source: Scientific American

Research into natural chemicals that mimic marijuana's effects in the brain could help to explain--and suggest treatments for--pain, anxiety, eating disorders, phobias and other conditions
By Roger A. Nicoll and Bradley N. Alger

Marijuana is a drug with a mixed history. Mention it to one person, and it will conjure images of potheads lost in a spaced-out stupor. To another, it may represent relaxation, a slowing down of modern madness. To yet another, marijuana means hope for cancer patients suffering from the debilitating nausea of chemotherapy, or it is the promise of relief from chronic pain. The drug is all these things and more, for its history is a long one, spanning millennia and continents. It is also something everyone is familiar with, whether they know it or not. Everyone grows a form of the drug, regardless of their political leanings or recreational proclivities. That is because the brain makes its own marijuana, natural compounds called endocannabinoids (after the plant's formal name, Cannabis sativa).

The study of endocannabinoids in recent years has led to exciting discoveries. By examining these substances, researchers have exposed an entirely new signaling system in the brain: a way that nerve cells communicate that no one anticipated even 15 years ago. Fully understanding this signaling system could have far-reaching implications. The details appear to hold a key to devising treatments for anxiety, pain, nausea, obesity, brain injury and many other medical problems. Ultimately such treatments could be tailored precisely so that they would not initiate the unwanted side effects produced by marijuana itself.

A Checkered Past
Marijuana and its various alter egos, such as bhang and hashish, are among the most widely used psychoactive drugs in the world. How the plant has been used varies by culture. The ancient Chinese knew of marijuana's pain-relieving and mind-altering effects, yet it was not widely employed for its psychoactive properties; instead it was cultivated as hemp for the manufacture of rope and fabric. Likewise, the ancient Greeks and Romans used hemp to make rope and sails. In some other places, however, marijuana's intoxicating properties became important. In India, for example, the plant was incorporated into religious rituals. During the Middle Ages, its use was common in Arab lands; in 15th-century Iraq it was used to treat epilepsy; in Egypt it was primarily consumed as an inebriant. After Napoleon's occupation of Egypt, Europeans began using the drug as an intoxicant. During the slave trade, it was transported from Africa to Mexico, the Caribbean and South America.

Marijuana gained a following in the U.S. only relatively recently. During the second half of the 19th century and the beginning of the 20th, cannabis was freely available without a prescription for a wide range of ailments, including migraine and ulcers. Immigrants from Mexico introduced it as a recreational drug to New Orleans and other large cities, where it became popular among jazz musicians. By the 1930s it had fallen into disrepute, and an intense lobbying campaign demonized "reefer madness." In 1937 the U.S. Congress, against the advice of the American Medical Association, passed the Marijuana Tax Act, effectively banning use of the drug by making it expensive and difficult to obtain. Ever since, marijuana has remained one of the most controversial drugs in American society. Despite efforts to change its status, it remains federally classified as a Schedule 1 drug, along with heroin and LSD, considered dangerous and without utility.

Millions of people smoke or ingest marijuana for its intoxicating effects, which are subjective and often described as resembling an alcoholic "high." It is estimated that approximately 30 percent of the U.S. population older than 12 have tried marijuana, but only about 5 percent are current users. Large doses cause hallucinations in some individuals but simply trigger sleep in others. The weed impairs short-term memory and cognition and adversely affects motor coordination, although these setbacks seem to be reversible once the drug has been purged from the body. Smoking marijuana also poses health risks that resemble those of smoking tobacco.

On the other hand, the drug has clear medicinal benefits. Marijuana alleviates pain and anxiety. It can prevent the death of injured neurons. It suppresses vomiting and enhances appetite--useful features for patients suffering the severe weight loss that can result from chemotherapy.

Finding the Responsible Agent
Figuring out how the drug exerts these myriad effects has taken a long time. In 1964, after nearly a century of work by many individuals, Raphael Mechoulam of the Hebrew University in Jerusalem identified delta-9-tetrahydrocannabinol (THC) as the compound that accounts for virtually all the pharmacological activity of marijuana. The next step was to identify the receptor or receptors to which THC was binding.

Receptors are small proteins embedded in the membranes of all cells, including neurons, and when specific molecules bind to them--fitting like one puzzle piece into another--changes in the cell occur. Some receptors have water-filled pores or channels that permit chemical ions to pass into or out of the cell. These kinds of receptors work by changing the relative voltage inside and outside the cell. Other receptors are not channels but are coupled to specialized proteins called G-proteins. These G-protein-coupled receptors represent a large family that set in motion a variety of biochemical signaling cascades within cells, often resulting in changes in ion channels.

In 1988 Allyn C. Howlett and her colleagues at St. Louis University attached a radioactive tag to a chemical derivative of THC and watched where the compound went in rats' brains. They discovered that it attached itself to what came to be called the cannabinoid receptor, also known as CB1. Based on this finding and on work by Miles Herkenham of the National Institutes of Health, Lisa Matsuda, also at the NIH, cloned the CB1 receptor. The importance of CB1 in the action of THC was proved when two researchers working independently--Catherine Ledent of the Free University of Brussels and Andreas Zimmer of the Laboratory of Molecular Neurobiology at the University of Bonn--bred mice that lacked this receptor. Both investigators found that THC had virtually no effect when administered to such a mouse: the compound had nowhere to bind and hence could not trigger any activity. (Another cannabinoid receptor, CB2, was later discovered; it operates only outside the brain and spinal cord and is involved with the immune system.)

INDIAN FAKIRS prepare bhang and ganja in this painting from the mid- 1700s. The history of marijuana extends far back in history, with written records on its medical use appearing in ancient Chinese and Egyptian texts. Discovery in the 1960s of its active component, THC, eventually led to identification of the brain's own "marijuana."
As researchers continued to study CB1, they learned that it was one of the most abundant G-protein coupled receptors in the brain. It has its highest densities in the cerebral cortex, hippocampus, hypothalamus, cerebellum, basal ganglia, brain stem, spinal cord and amygdala. This distribution explains marijuana's diverse effects. Its psychoactive power comes from its action in the cerebral cortex. Memory impairment is rooted in the hippocampus, a structure essential for memory formation. The drug causes motor dysfunction by acting on movement control centers of the brain. In the brain stem and spinal cord, it brings about the reduction of pain; the brain stem also controls the vomiting reflex. The hypothalamus is involved in appetite, the amygdala in emotional responses. Marijuana clearly does so much because it acts everywhere.

Over time, details about CB1's neuronal location emerged as well. Elegant studies by Tamás F. Freund of the Institute of Experimental Medicine at the Hungarian Academy of Sciences in Budapest and Kenneth P. Mackie of the University of Washington revealed that the cannabinoid receptor occurred only on certain neurons and in very specific positions on those neurons. It was densely packed on neurons that released GABA (gamma-aminobutyric acid), which is the brain's main inhibitory neurotransmitter (it tells recipient neurons to stop firing). CB1 also sat near the synapse, the contact point between two neurons. This placement suggested that the cannabinoid receptor was somehow involved with signal transmission across GABA-using synapses. But why would the brain's signaling system include a receptor for something produced by a plant?

The Lesson of Opium
The same question had arisen in the 1970s about morphine, a compound isolated from the poppy and found to bind to so-called opiate receptors in the brain. Scientists finally discovered that people make their own opioids--the enkephalins and endorphins. Morphine simply hijacks the receptors for the brain's opioids.

It seemed likely that something similar was happening with THC and the cannabinoid receptor. In 1992, 28 years after he identified THC, Mechoulam discovered a small fatty acid produced in the brain that binds to CB1 and that mimics all the activities of marijuana. He named it anandamide, after the Sanskrit word ananda, "bliss." Subsequently, Daniele Piomelli and Nephi Stella of the University of California at Irvine discovered that another lipid, 2-arachidonoyl glycerol (2-AG), is even more abundant in certain brain regions than anandamide is. Together the two compounds are considered the major endogenous cannabinoids, or endocannabinoids. (Recently investigators have identified what look like other endogenous cannabinoids, but their roles are uncertain.) The two cannabinoid receptors clearly evolved along with endocannabinoids as part of natural cellular communication systems. Marijuana happens to resemble the endocannabinoids enough to activate cannabinoid receptors.

Conventional neurotransmitters are water-soluble and are stored in high concentrations in little packets, or vesicles, as they wait to be released by a neuron. When a neuron fires, sending an electrical signal down its axon to its tips (presynaptic terminals), neurotransmitters released from vesicles cross a tiny intercellular space (the synaptic cleft) to receptors on the surface of a recipient, or postsynaptic, neuron. In contrast, endocannabinoids are fats and are not stored but rather are rapidly synthesized from components of the cell membrane. They are then released from places all over the cells when levels of calcium rise inside the neuron or when certain G-protein-coupled receptors are activated.

As unconventional neurotransmitters, canna-bin-oids presented a mystery, and for several years no one could figure out what role they played in the brain. Then, in the early 1990s, the answer emerged in a somewhat roundabout fashion. Scientists (including one of us, Alger, and his colleague at the University of Maryland School of Medicine, Thomas A. Pitler) found something unusual when studying pyramidal neurons, the principal cells of the hippocampus. After calcium concentrations inside the cells rose for a short time, incoming inhibitory signals in the form of GABA arriving from other neurons declined.

At the same time, Alain Marty, now at the Laboratory of Brain Physiology at the René Descartes University in Paris, and his colleagues saw the same action in nerve cells from the cerebellum. These were unexpected observations, because they suggested that receiving cells were somehow affecting transmitting cells and, as far as anyone knew, signals in mature brains flowed across synapses in one way only: from the presynaptic cell to the postsynaptic one.

A New Signaling System
it seemed possible that a new kind of neuronal communication had been discovered, and so researchers set out to understand this phenomenon. They dubbed the new activity DSI, for depolarization-induced suppression of inhibition. For DSI to have occurred, some unknown messenger must have traveled from the postsynaptic cell to the presynaptic GABA-releasing one and somehow shut off the neurotransmitter's release.

Such backward, or "retrograde," signaling was known to occur only during the development of the nervous system. If it were also involved in interactions among adult neurons, that would be an intriguing finding--a sign that perhaps other processes in the brain involved retrograde transmission as well. Retrograde signaling might facilitate types of neuronal information processing that were difficult or impossible to accomplish with conventional synaptic transmission. Therefore, it was important to learn the properties of the retrograde signal. Yet its identity remained elusive. Over the years, countless molecules were proposed. None of them worked as predicted.

Then, in 2001, one of us (Nicoll) and his colleague at the University of California at San Francisco, Rachel I. Wilson--and at the same time, but independently, a group led by Masanobu Kano of Kanazawa University in Japan--reported that an endocannabinoid, probably 2-AG, perfectly fit the criteria for the unknown messenger. Both groups found that a drug blocking cannabinoid receptors on presynaptic cells prevents DSI and, conversely, that drugs activating CB1 mimic DSI. They soon showed, as did others, that mice lacking cannabinoid receptors are incapable of generating DSI. The fact that the receptors are located on the presynaptic terminals of GABA neurons now made perfect sense. The receptors were poised to detect and respond to endocannabinoids released from the membranes of nearby postsynaptic cells.

Over time, DSI proved to be an important aspect of brain activity. Temporarily dampening inhibition enhances a form of learning called long-term potentiation--the process by which information is stored through the strengthening of synapses. Such storage and information transfer often involves small groups of neurons rather than large neuronal populations, and endocannabinoids are well suited to acting on these small assemblages. As fat-soluble molecules, they do not diffuse over great distances in the watery extracellular environment of the brain. Avid uptake and degradation mechanisms help to ensure that they act in a confined space for a limited period. Thus, DSI, which is a short-lived local effect, enables individual neurons to disconnect briefly from their neighbors and encode information.

A host of other findings filled in additional gaps in understanding about the cellular function of endocannabinoids. Researchers showed that when these neurotransmitters lock onto CB1 they can in some cases block presynaptic cells from releasing excitatory neurotransmitters. As Wade G. Regehr of Harvard University and Anatol C. Kreitzer, now at Stanford University, found in the cerebellum, endocannabinoids located on excitatory nerve terminals aid in the regulation of the massive numbers of synapses involved in coordinated motor control and sensory integration. This involvement explains, in part, the slight motor dysfunction and altered sensory perceptions often associated with smoking marijuana.

Recent discoveries have also begun to precisely link the neuronal effects of endocannabinoids to their behavioral and physiological effects. Scientists investigating the basis of anxiety commonly begin by training rodents to associate a particular signal with something that frightens them. They often administer a brief mild shock to the feet at the same time that they generate a sound. After a while the animal will freeze in anticipation of the shock if it hears the sound. If the sound is repeatedly played without the shock, however, the animal stops being afraid when it hears the sound--that is, it unlearns the fear conditioning, a process called extinction. In 2003 Giovanni Marsicano of the Max Planck Institute of Psychiatry in Munich and his co-workers showed that mice lacking normal CB1 readily learn to fear the shock-related sound, but in contrast to animals with intact CB1, they fail to lose their fear of the sound when it stops being coupled with the shock.

The results indicate that endocannabinoids are important in extinguishing the bad feelings and pain triggered by reminders of past experiences. The discoveries raise the possibility that abnormally low numbers of cannabinoid receptors or the faulty release of endogenous cannabinoids are involved in post-traumatic stress syndrome, phobias and certain forms of chronic pain. This suggestion fits with the fact that some people smoke marijuana to decrease their anxiety. It is also conceivable, though far from proved, that chemical mimics of these natural substances could allow us to put the past behind us when signals that we have learned to associate with certain dangers no longer have meaning in the real world.

Devising New Therapies
The repertoire of the brain's own marijuana has not been fully revealed, but the insights about endocannabinoids have begun helping researchers design therapies to harness the medicinal properties of the plant. Several synthetic THC analogues are already commercially available, such as nabilone and dronabinol. They combat the nausea brought on by chemotherapy; dronabinol also stimulates appetite in AIDS patients. Other cannabinoids relieve pain in myriad illnesses and disorders. In addition, a CB1 antagonist--a compound that blocks the receptor and renders it impotent--has worked in some clinical trials to treat obesity. But though promising, these drugs all have multiple effects because they are not specific to the region that needs to be targeted. Instead they go everywhere, causing such adverse reactions as dizziness, sleepiness, problems of concentration and thinking abnormalities.

One way around these problems is to enhance the role of the body's own endocannabinoids. If this strategy is successful, endocannabinoids could be called forth only under the circumstances and in the locations in which they are needed, thus avoiding the risks associated with widespread and indiscriminant activation of cannabinoid receptors. To do this, Piomelli and his colleagues are developing drugs that prevent the endocannabinoid anandamide from being degraded after it is released from cells. Because it is no longer broken down quickly, its anxiety-relieving effects last longer.

Anandamide seems to be the most abundant endocannabinoid in some brain regions, whereas 2-AG dominates in others. A better understanding of the chemical pathways that produce each endocannabinoid could lead to drugs that would affect only one or the other. In addition, we know that endocannabinoids are not produced when neurons fire just once but only when they fire five or even 10 times in a row. Drugs could be developed that would alter the firing rate and hence endocannabinoid release. A precedent for this idea is the class of anticonvulsant agents that suppress the neuronal hyperactivity underlying epileptic seizures but do not affect normal activity.

Finally, indirect approaches could target processes that themselves regulate endocannabinoids. Dopamine is well known as the neurotransmitter lost in Parkinson's disease, but it is also a key player in the brain's reward systems. Many pleasurable or addictive drugs, including nicotine and morphine, produce their effects in part by causing dopamine to be released in several brain centers. It turns out that dopamine can cause the release of endocannabinoids, and various research teams have found that two other neurotransmitters, glutamate and acetylcholine, also initiate endocannabinoid synthesis and release. Indeed, endocannabinoids may be a source of effects previously attributed solely to these neurotransmitters. Rather than targeting the endocannabinoid system directly, drugs could be designed to affect the conventional neurotransmitters. Regional differences in neurotransmitter systems could be exploited to ensure that endocannabinoids would be released only where they were needed and in appropriate amounts.

In a remarkable way, the effects of marijuana have led to the still unfolding story of the endocannabinoids. The receptor CB1 seems to be present in all vertebrate species, suggesting that systems employing the brain's own marijuana have been in existence for about 500 million years. During that time, endocannabinoids have been adapted to serve numerous, often subtle, functions. We have learned that they do not affect the development of fear, but the forgetting of fear; they do not alter the ability to eat, but the desirability of the food, and so on. Their presence in parts of the brain associated with complex motor behavior, cognition, learning and memory implies that much remains to be discovered about the uses to which evolution has put these interesting messengers.


ROGER A. NICOLL and BRADLEY E. ALGER first worked together in the late 1970s, when they both were forming what has become an enduring interest in synaptic transmission. At that time, Nicoll had just moved to the University of California, San Francisco, where he is now professor of pharmacology; Alger, currently professor of physiology and psychiatry at the University of Maryland School of Medicine, was his first postdoctoral fellow. Nicoll is a member of the National Academy of Sciences and recent winner of the Heinrich Wieland Award

Also on high times

http://hightimes.com/ht/legal/content.php?bid=128&aid=3

HOW TO EXERCISE YOUR LEGAL RIGHTS
2002-09-20 >> legal category >> legal article

The United States Constitution provides the foundation for the rights that protect all U.S. citizens from intrusive law enforcement practices. These rights should be exercised by everyone in all circumstances, regardless of whether or not an individual is guilty of a crime. Rights are like muscles -- if they are not exercised, they wither away.

RULES OF THUMB

Never leave anything in plain view: Although law enforcement officers must obtain a warrant before they can conduct a privacy-invading search, any illicit material that can be plainly seen by any person from a non-intrusive vantage point is subject to confiscation. An arrest and a valid warrant to search the rest of the area is likely to ensue. A "roach" in the ashtray, a pipe or baggie on the dashboard or coffee table, or a joint being smoked in public are common mistakes that can lead to prosecution.

Never put anything incriminating into the trash: Various courts have ruled that law enforcement officers are allowed to rummage through curbside trash bags without a warrant. A few seeds or stems can then be used as a basis for obtaining a warrant to search the individual's home.
In fact, anything discarded into the public domain can be picked up by the police and used as evidence. For example, if an individual throws an illicit substance out of his or her car window and a police officer sees it and picks it up, the person is almost certain to be arrested.

NEVER CONSENT TO A SEARCH: Most individuals arrested on marijuana charges could have avoided the arrest by exercising their Fourth Amendment rights. If a law enforcement officer asks permission to search, it is usually because: (1) there is not enough evidence to obtain a search warrant; or (2) the officer does not feel like going through the hassle of obtaining a warrant.

Law enforcement officers are trained to intimidate people into consenting to searches. If an individual does consent, the officer can -- and will -- conduct the search without a warrant. If the officer finds any contraband, the person will be arrested. Moreover, the validity of the evidence will almost definitely hold up in court because consenting to a search essentially amounts to handing the evidence to the officer and saying, "Here it is -- arrest me."

If an individual does not consent, the officer must either release the person or detain the person and attempt to get a warrant. The fact that an individual refuses to consent does not give the officer grounds to obtain a warrant. The individual should politely say:

"I do not consent to a search of my person, belongings, home, or vehicle. I retain my Fourth Amendment rights and all other rights under the United States Constitution. I will say nothing until my attorney is present."

If the officer conducts a search anyway --without a warrant-- any contraband will likely be declared invalid evidence by the judge, and any charges will probably be dropped. If the officer does attempt to get a warrant and is successful in doing so, any contraband discovered may still be excluded as evidence if the individual's lawyer can convince the judge that the warrant itself was invalid --which, in many cases, it is. No matter what a law enforcement officer threatens or promises, it is always better to refuse to consent to a search.

EXERCISE YOUR RIGHTS [cont.]

LOOSE LIPS SINK SHIPS: Whether arrested or not, individuals should always exercise the right to remain silent. Anything a person says to law enforcement officers, reporters, cellmates, or even their friends can--and probably will--be used as evidence against them.

Individuals have the right to have an attorney present during questioning. Only a qualified attorney can ensure that the suspect or defendant does not say anything damaging. The right to remain silent should always be exercised; three hypothetical examples follow:

--Cop: "Is this your pipe?"
--Joe Citizen: "My attorney advised me to remain silent unless she is present."

--Cop: "If I look in your trunk, I'm not going to find any drugs?"
--Jane Citizen: "I do not consent to a search of my trunk, and I'd rather not answer any questions without an attorney present."

--Cop: [during search of apartment -- with a warrant-- upon discovering and examining a tiny bag of leaves and stems]: "Looks like a couple pounds of good bud here ... too bad. You can do some serious time in the state slammer for this."
--John Citizen: [says nothing at all]

DO NOT STICK AROUND ANY LONGER THAN IS REQUIRED: From the time a law enforcement officer approaches, it is wise to remain calm and not arouse suspicion. Nevertheless, individuals should always find out if the officer requires them to stay; if not, they should explain that they are in a hurry, then leave.

Law enforcement officers are trained to create the impression that their suspects are obliged to stay. Individuals being questioned by an officer should simply say: "Am I under arrest or otherwise detained? If not, I really need to get going. Have a nice day."

DO NOT BE HOSTILE; DO NOT PHYSICALLY RESIST: Some law enforcement officers do not care about citizens' rights; sometimes, the suspect is caught red-handed; other times, there are special-case qualifiers to certain rights, or there are loopholes beyond the scope of discussion in this publication. In any case, there are times when individuals politely assert their rights and refuse to talk or give consent, but the officers disregard their wishes and proceed to detain, search, or arrest them. In such cases, it is important to keep in mind that law enforcement officers have clubs, mace, handcuffs, guns, back-up, and usually the trust of the court. Aggression against the officers can make matters far worse. This does not mean that individuals facing such circumstances should give up all rights. Sometimes it is best to simply say, "Do what you feel you must; I will not physically resist. However, I do not consent to this."

DO NOT BE A SNITCH: The police and prosecutors often try to pressure individuals into providing information that would lead to the arrest and conviction of others. Sometimes a person's own defense attorney will even encourage him or her to comply!

A wise marijuana consumer will avoid the issue entirely by reducing the possibility of apprehension by knowing his or her rights. However, prudent marijuana consumers will keep in mind that the possibility of arrest always exists. They remember the adage: "Don't do the crime if you can't do the time."

Threats and promises by police and prosecutors should be viewed with caution and skepticism. Decisions should only be made after consulting with an attorney and examining one's own conscience. Just remember, saving one's self by pointing the finger at others is the most cowardly thing a person can do. There is no justification for being a traitor in the War Against Marijuana Consumers.

damn, dude go write at a fucking newspaper. shit i can`t even grasp that shiat!!!

Howdy Smoke,

Thanx for the interesting articles..I found the first article to be fascinating..and the second- full of common sense and advice. I especially liked the last paragraph of the first article :

" In a remarkable way, the effects of marijuana have led to the still unfolding story of the endocannabinoids. The receptor CB1 seems to be present in all vertebrate species, suggesting that systems employing the brain's own marijuana have been in existence for about 500 million years. During that time, endocannabinoids have been adapted to serve numerous, often subtle, functions. We have learned that they do not affect the development of fear, but the forgetting of fear; they do not alter the ability to eat, but the desirability of the food, and so on. Their presence in parts of the brain associated with complex motor behavior, cognition, learning and memory implies that much remains to be discovered about the uses to which evolution has put these interesting messengers. "

I think,that this paragraph can be summed up like this :

The Lord..works in mysterious ways .. I believe that marijuana is one of God's most perfect creations,it's molecules are complex and their effects,multi-fold and still somewhat mysterious in scope. That it is capable of effecting the forgetting of fear..ranging to desirability of food,is miraculous to me..and proof that only a Divine Creator..could have had the vision to bless the human species with such a miraculous plant.

I have come to believe,that the answer to all of Man's ills,can be found in Nature..if only we can find such and learn it's uses. I also believe,that the CB1 and CB2 receptors,were designed by God to facillitate complex healing and protection measures,as well as,connect us with our spiritual selves,opening and expanding our consciences to the spiritual world. Simply put-when I'm stoned..I feel closer to God and His Heavenly Kingdom..I'm more at tune with Nature..and I can make global connections of thought,that I cannot make-when I'm not stoned.

Well..I've rambled on long enough..I hope that I haven't offended you or anyone with my opinion..if so..git stoned..you'll git over it ! lol

Have a good one....Torog :D

wow

just wow


great advice for dealing with the police, though! most people don't know that cops don't automatically have the right to search you.

I think I should add though, the bit aboot "--Cop: [during search of apartment -- with a warrant-- upon discovering and examining a tiny bag of leaves and stems]: "Looks like a couple pounds of good bud here ... too bad. You can do some serious time in the state slammer for this."
--John Citizen: [says nothing at all] "
is this silence so that John Citizen is not held liable for admission of possession of 'a couple pounds of good bud' like the good officer said (even though it is clearly false)?

I hate the U.S. government so much.

Quote:

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Originally Posted by IntrepidS

I hate the U.S. government so much.

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Hating the government is about as American as it gets ;)


Great write ups man, very interesting reading (the first one) and very refreshing read (2nd one).

yeah i liked it. bit long but got through it in the end :) good advise bout police have to remember that "my atterney advised me to remain silent until he/she is present" not that i have 1!!
But a good find :)

thanks guys thats what happens when i do speed god dmn my friends need to stay away from me some times! and thank god for spell cheac!! HAAHAH


yes do not forget if a cp ask you if he can such your house or you car OR YOU! even if you dont have anything to hide!!! the answer is ALWAYS! do you have a warrent? also SLIENT!! if you DO NOT SAY IT they cant use it you can nod up and down all you want ((so i understand it)) as long as you dont say "yes" they cant do anything with out a warrent therefore giveing you lots of time to get rid of all evadence.


even if you dont have an atterney. you can demade to speak to one. BY LAW they must appoint one to you its your rights as an american


i was wandering though can i relly get my dog high? i mean i know they get hungery as hell but do they "feel" high? i am looking for studies done on dogs to see if they have the same receptors.

**scrolls up* holy shit! ahh screw that it would take me 30 mins to correct all that with spell check! what the fuck is "spell cheac????????" LOL

Quote:

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Originally Posted by IntrepidS

I hate the U.S. government so much.

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its good to see you've joined the view of the rest of the world.